Figure 5
From: Regulation of lung oxidative damage by endogenous superoxide dismutase in sepsis
Inducible nitric oxide synthase (iNOS) alterations early after sepsis induction. The animals were submitted to sepsis or sham-operated, and 3 h (A), 6 h (B), and 12 h (C) after surgery, the immunocontent of iNOS was evaluated in the lung and in the pulmonary artery (3 h (D), 6 h (E), 12 h (F)). The data are expressed as mean ± SD (n = ten animals/group). p value < 0.05 was considered significant when compared to the control.