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Table 1 Physiological data

From: Cardiovascular disease and resuscitated septic shock lead to the downregulation of the H2S-producing enzyme cystathionine-γ-lyase in the porcine coronary artery

   Baseline 24 h
NoA (μg/(kg min)) Sham 0.06 (0.02; 0.13)
Sepsis 1.23 (0.66; 3.26)a
Mean arterial pressure (mmHg) Sham 100 (90; 106) 103 (94; 119)
Sepsis 103 (91; 112) 65 (61; 81)a, b
Central venous pressure (mmHg) Sham 8 (7; 13) 10 (9; 15)b
Sepsis 10 (6; 13) 17 (14; 18)b
Cardiac output (ml/(kg min)) Sham 61 (52; 79) 64 (42; 92)
Sepsis 64 (52; 69) 87 (62; 130)b
pO2 (mmHg) Sham 158 (142; 180) 159 (138;177)
Sepsis 170 (161; 183) 93 (62; 155)a, b
pCO2 (mmHg) Sham 35 (35; 39) 35 (33; 36)
Sepsis 38 (34; 40) 35 (32; 44)
arterial pH Sham 7.46 (7.44; 7.46) 7.44 (7.43; 7.46)
Sepsis 7.45 (7.43; 7.48) 7.37 (7.19; 7.43)a, b
Lactate (mmol/l) Sham 1.4 (1.0; 1.6) 0.6 (0.6; 1.2)
Sepsis 0.8 (0.6; 1.5) 6.1 (2.0; 10.7)a, b
Base excess (mmol/l) Sham 1.1 (0.8; 1.8) −0.1(−1.45; 0.65)
Sepsis 1.5 (0.4; 2.3) −8.5 (−14.6; −3.7)a, b
  1. Physiological data, presented as median (interquartile range, IQR). Sham n = 5, sepsis n = 8
  2. NoA norepinephrine infusion rate, pO 2 partial oxygen pressure, pCO 2 partial carbon dioxide pressure
  3. aSignificant compared to sham
  4. bSignificant compared to baseline