Fig. 2From: Molecular mechanisms of postintensive care syndromeMechanisms of chronic lung dysfunction after critical illness. Local inflammation, changes in cell populations and matrix remodeling promote a pro-fibrotic state that may cause long-term respiratory impairment. TRPV4 transient receptor potential cation channel, subfamily V, member 4. BBB blood–brain barrier, DRD2 dopamine receptor type 2Back to article page