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Intensive Care Medicine Experimental
Fig. 3 | Intensive Care Medicine Experimental

Fig. 3

From: Clinical phenotypes and outcomes associated with SARS-CoV-2 Omicron variants BA.2, BA.5 and BQ.1.1 in critically ill patients with COVID-19: a prospective, multicenter cohort study

Fig. 3

Prevalence of amino acid substitutions and deletions in Spike RBD in BA.2, BA.4/BA.5, and BQ.1.1-related group viral sequences; The percentage of detected mutations (amino acid substitutions and deletions) per group is displayed on the y-axis, relative to the original Omicron BA.2 reference sequence (SARS-CoV-2/human/USA/FL-CDC-STM-77CPCCUR3/2022). Other individual NTD (N-terminal domain) mutations include H49Y, W64R, M153I, M177L, I197T, Del211, L212I, A222S, T250I, P251H, V289I and S316F; other S1/RBD mutations include N354K, I358L, T376S, T547K, T572I and N568S; other S2 mutations include T547K, T572I, H625R, A642G, A647V, E654K, N658S, N856S, V963F, A1020S, T1117I, P1143L, E1144Q and S1249P

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