Fig. 1From: Scavenging reactive oxygen species using tempol in the acute phase of renal ischemia/reperfusion and its effects on kidney oxygenation and nitric oxide levelsRenal oxygenation, oxidative stress, and nitric oxide (NO) levels at the end of the protocol. a Microvascular oxygen tensions (μpO2) in the renal cortex; b Microvascular oxygen tensions (μpO2) in the renal medulla; c renal tissue malondialdehyde (MDA) levels normalized to the tissue protein content; and d tissue NO levels normalized to the tissue protein content. *p < 0.05 vs all other groups; C p < 0.05 vs the CTRL group; T p < 0.05 vs the TMPL groupBack to article page