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Erratum to: Heparin-binding protein is important for vascular leak in sepsis

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The original article was published in Intensive Care Medicine Experimental 2016 4:33

Erratum

The original article [1] contains an oversight whereby Fig. 7 presents data on a molecule, ‘PLAH’ that was intended to be omitted from this article.

The reason for the intended omission of this molecule throughout the article was due to complications in obtaining the license for both the use of and presentation of research involving this molecule, and its presence within Fig. 7 was unintentional.

As such, the appropriate version of Fig. 7 can be seen below.

Fig. 7
figure1

Low molecular weight heparins blocked HBP-induced permeability increases. EA.hy926 cells were grown to confluence on permeable supports and stimulated with 10 μg/mL HBP, pre-incubated with the indicated inhibitor. TEER was measured after 1.5 h after stimulation and is normalized to empty inserts. Error bars are standard error of the mean, n = 3 for each condition. One-way ANOVA with Dunnett’s test for multiple comparisons was used to compare each group to the condition with HBP and no inhibitor (far left). UFH unfractionated heparin. *P < 0.05, **P < 0.01

Reference

  1. 1.

    Bentzer P et al (2016) Heparin-binding protein is important for vascular leak in sepsis. Intensive Care Medicine Experimental 4:33. doi:10.1186/s40635-016-0104-3

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Author information

Correspondence to Adam Linder.

Additional information

The online version of the original article can be found under doi:10.1186/s40635-016-0104-3.

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